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The accumulation of AEGs and their binding to the receptor for glycosylation end-products (RAGE) promotes oxidative stress, which activates the NF-κB signalling pathway, upregulates the expression of target genes and triggers inflammation (22)

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3d), however, this is in the context of the CT-AMY 2 R complex adopting a “CT” conformation, compared to the “bypass” conformation stabilised by Cagri