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However, in a subanalysis of participants with low vitamin B 12 status at baseline (n= 101), the B-vitamin supplementation slowed the decline in bone mineral density (total hip and femoral neck but not lumbar spine) (116)

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To evaluate the target-binding capacity of D-CuP, molecular docking simulations were conducted using AutoDock Vina against the functional domains of p53 (DNA-binding domain) and HDAC7 (catalytic deacetylase domain)