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Its mechanisms include: Actin polymerization — mobilizes cellular building blocks for tissue repair Cell migration — accelerates fibroblast and keratinocyte movement to injury sites Angiogenesis — promotes new blood vessel formation via VEGF upregulation Anti-inflammatory — reduces IL-1β and TNF-α at injury sites TB-500's smaller molecular size gives it excellent tissue distribution throughout the body
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NO-system immobilization (L-NAME+L-arginine)) reveals that each of „negative symptoms“ differs from each other given their different responsibility to L-NAME and L-arginine given alone or together
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